Acidification and disease
An increasing number of scientists around the world agree that the acidification of the body is the source of all disease. When the body cannot cope with the build-up of acidic waste and resist the damaging effect of free radicals, we become sick. This usually occurs around the age of 40-45. Depending on its area of accumulation, symptoms of a specific disease may develop. The accumulation of acids in the area of the pancreas disturbs the secretion of insulin, which may lead to diabetes.
- Type I diabetes is caused by an insulin deficiency due to the oxidative damage of pancreatic beta cells. Beta cells are found in clusters of pancreatic cells known as Langerhans islets. They are responsible for insulin secretion. Insulin regulates the metabolic processing of protein, fat and especially carbohydrates. It promotes the transportation of glucose from the blood stream to the cells. Diabetes occurs when the secretion of insulin is disturbed and the cells are unable to take in the glucose that is indispensable in their vital activity. Barred from making its way to the cells, glucose concentrations accumulate in the blood. As a result, the cells are forced to starve and blood-glucose levels increase significantly.
- Type II diabetes is also strongly associated with the oxidative damage of myotube and adipocite cells due to stress, excessive eating and lack of exercise. In this case the body is not dependent on insulin production but it is incapable of using it properly. This is called insulin resistance. At first, the pancreas increases insulin production to stimulate the absorption of glucose accumulating in the blood. But when the beta cell resources responsible for insulin production are depleted, insulin production slows down. This form of disease generally affects overweight and older people in their 40s. Due to this excess of glucose in the blood, such patients have a distinctly more pronounced appetite and, unfortunately, excess weight.
Modern medicine has still not cured diabetes. Throughout their lives, diabetes patients must take oral or injectable medication to help normalize the glucose levels in their blood. However, some research shows that regular use of alkaline ionized water may help maintain normal blood-sugar levels and reduce the amount of insulin that needs to be injected. Scientists in Germany, Japan and Korea have investigated the effect of alkaline water on diabetes patients and described various success stories, including with diabetes treatment. Their findings are presented below.
Excess glucose will accumulate in the blood and bind to haemoglobin. Glucose and protein compounds accumulating in blood vessels will jam up capillaries, leading to the development of atherosclerosis. When circulation is impeded, tissues don’t receive an adequate supply of nutrients and diabetes-related complications start to develop.
On the one hand, the calcium ions received with alkaline water hinder the formation of glucose and protein compounds. On the other, they neutralize the accumulation of acidic waste around the pancreas, ensuring improved pancreatic function. Most importantly, the consumption of alkaline water – a natural antioxidant – ensures the protection of pancreatic beta-cells against the disruptive effects of free radicals.
A clinic in Berlin published highly promising clinical research results in the treatment of type I and type II diabetes. Researcher Dr. Dina Aschbach administered alkaline waterto her patients along with regular treatment. The water was supplemented with additional quantities of several trace elements, primarily calcium ions. In a period of four weeks, the level of glycated haemoglobin dropped to 2 percentage points, the demand for insulin injection – to 70 percent, and glucose blood levels – to 30 percent. These numbers continued dropping for another 4-5 months even after the patients stopped drinking alkaline water.
Similar results have been produced for type I diabetes patients. A decreased demand for insulin in cases of type I diabetes is based on the assumption that a part of pancreatic beta cells are asleep. The negative potential of alkaline water wakes them up, thus stimulating their activity (Aschbach, 2010).
According to a national health and nutrition survey conducted in 2007, 22.1 million people in Japan – a staggering sixth of the country’s population – have been diagnosed with diabetes or are suspected to have it (Shirahata et al., 2012). In response to this dangerous trend, several groups of Japanese researchers carried out studies to figure out the impact of alkaline water, otherwise known as electrolyzed reduced water (ERW) on diabetes patients.
Since the 1990s, electrolyzed reduced water has been attracting increasingly more attention because it has been shown to possess antioxidative effects by functioning as a free-radical scavenger. ERW scavenged ROS (free radicals derived from molecular oxygen) in vitro and protected DNA from oxidative damage (Shirahata et al. 1997), stimulating glucose intake by muscle cells and adipocytes (Oda et al. 1999).
Several years later, ERW water was shown to protect pancreatic beta cells from cell damage induced by alloxan, a diabetogenic compound associated with type I diabetes. Its diabetogenic effect is exerted via the production of ROS. Research results suggest that reduced water protects pancreatic beta-cells from alloxan-induced cell damage by preventing alloxan-derived ROS generation. The study concluded that reduced water may be useful in preventing alloxan-induced type 1-diabetes mellitus (Li et al., 2002).
A group of Korean researchers conducted several studies examining the possible anti-diabetic effects of electrolyzed reduced water (ERW) in diabetic animals.
The effects are based on the antioxidant properties of ERW – i.e. the ability to scavenge ROS. One study has shown that ERW, provided as a drinking water, significantly reduced the blood glucose concentration and improved glucose tolerance in both type I and type II diabetic animal models. The data suggested that ERW could function as an orally effective anti-diabetic agent and merit further study of its precise mechanism (Kim & Kim, 2006).
Another study examined the possible anti-diabetic effect of ERW in genetically diabetic mice. With its ROS scavenging ability, ERW reduced blood glucose concentration, increased blood insulin levels, improved glucose tolerance and preserved beta-cell mass in diabetic mice. The data suggested that ERW could protect beta-cell damage and would be useful as an anti-diabetic agent (Kim et al., 2007).
- Dr. Dina Aschbach. Ionisiertes Wasser – die moderne Medizin unserer Zeit, Hochheim 2010.
- Shirahata S., Hamasaki T., Teruya K. Advanced research on the health benefit of reduced water. Trends in Food Science and Technology 23 (2012), pp. 124-131
- Li Y., Nishimura T., Teruya K., Maki T., Komatsu T., Hamasaki T., Kashiwagi T., Kabayama S., Shim S., Katakura Y., Osada K., Kawahara T., Otsubo K., Morisawa S., Ishii Y., Gadek Z., Shirahata S. Protective mechanism of reduced water against alloxan-induced pancreatic beta-cell damage: Scavenging effect against reactive oxygen species. Cytotechnology. 2002 Nov; 40(1-3):139-49.
- Oda M., Kusumoto K., Teruya K., Hara T., Maki T., Kabayama Y., Katakura Y., Otsubo K., Morisawa S., Hayashi H., Ishii Y., Shirahata S., et al. Electrolyzed and natural reduced water exhibit insulin-like activity on glucose uptake into muscle cells and adipocytes. In: Bernard A, et al., editors. Animal cell technology: products from cells, cells as products. The Netherlands: Kluwer; 1999. pp. 425–427.
- Kim MJ, Kim HK. Anti-diabetic effects of electrolyzed reduced water in streptozotocin-induced and genetic diabetic mice. Life Sci. 2006 Nov 10; 79 (24): 2288-92.
- Kim MJ, Jung KH, Uhm YK, Leem KH, Kim HK. Preservative effect of electrolyzed reduced water on pancreatic beta-cell mass in diabetic db/db mice. Biol Pharm Bull. 2007 Feb; 30(2): 234-6.